Scientists have identified a new mechanism which contributes to the development of idiopathic pulmonary fibrosis (IPF).
They showed that the pathological changes of lung tissue are accompanied by an increase in protein turnover by the central protein degradation machinery of the cell – the proteasome. Their study has now been published in the American Journal of Respiratory and Critical Care Medicine.
Increased Protein Turnover Contributes to Development of Pulmonary Fibrosis
